Mélanie DesChâtelets

By Dr. Mélanie DesChâtelets, ND. Licensed naturopathic doctor and founder of PMOS Health Collective.

What PCOS type am I? If you’ve typed that into a search bar, you’re in good company. Adrenal, inflammatory, insulin-resistant, post-pill… maybe you see yourself in a few buckets at once and you’re trying to work out how to split yourself between them. Here’s the honest answer to “what PCOS type am I”: the popular online types aren’t medical diagnoses, and the real research types weren’t built to tell an individual what to do. Let me show you where the buckets come from, why they sound so legit, and what to use instead.

One quick note first. This is education, not medical advice, and I’m not your naturopathic doctor. Read it, take notes, then bring your own picture to your care team.

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“Type” means two different things

Here’s the first source of confusion. The word “type” gets used two completely different ways, and they get blurred together. One is the popular subtypes you’ve seen online (adrenal, inflammatory, insulin-resistant, post-pill). The other is the four research phenotypes (you might have seen them as A, B, C, or D). They are not the same thing. So when you ask “what type am I,” the honest first step is sorting out which kind of “type” you even mean.

Are the online subtypes a real diagnosis?

The popular subtypes come from mechanism-based theories, a way to explain the different reasons PCOS might show up in someone’s body. That instinct isn’t bad. But they aren’t medical diagnoses, they don’t appear in research papers, and they aren’t in the clinical guidelines. What happened next is the problem: the ideas got turned into fixed categories, and then protocols, supplement stacks, even specific diets and workouts got built around each bucket.

And here’s the truth, the reason so many people land here frustrated: PCOS doesn’t fit a neat bucket. You can be insulin-resistant and have inflammation. There’s so much overlap that people end up splitting themselves across buckets and merging protocols, and it gets messy. If you’ve always struggled to fit just one, congratulations, you figured out why this doesn’t work ahead of everyone else. The good news: a lot of the evidence-based suggestions help several “buckets” at once. Things get better together.

The four research phenotypes

Now the other meaning. Because PCOS is diagnosed on two of three criteria, it genuinely looks different person to person, and the phenotypes just describe which diagnostic features are present. This isn’t about mechanisms like insulin or inflammation, it’s about what’s actually on the diagnostic picture:

• Phenotype A: all three, irregular cycles, high androgens, and ovarian morphology (elevated AMH or ultrasound).
• Phenotype B: irregular cycles and high androgens, no ovarian morphology.
• Phenotype C: ovarian morphology and high androgens, but regular cycles.
• Phenotype D: irregular cycles and ovarian morphology, but no high-androgen symptoms.

These groupings are genuinely useful for studying large patterns across big populations, and I’m a proponent of that research. It may tell us whether certain groups carry higher or lower risk of certain things. That’s a step in the right direction.

Why a type can’t tell you what to do

Where it misses the mark is stretching population data onto the one person in front of us. Even if something is more likely in one phenotype, it doesn’t mean it can’t happen in another. So if one phenotype is “higher risk” for insulin resistance, you should still screen for insulin resistance in all the others, because the label isn’t a way to exclude it. That mirrors the PCOS guideline and what the brightest minds in PCOS care are saying: screen everyone, then individualize based on what’s actually showing up for you and what isn’t, because you checked. The mechanisms matter. Grouping people by them just isn’t as helpful as it sounds.

Where the labels backfire

Being told you’re “the adrenal type” or “the inflammatory type” can feel like a lightbulb moment. But what usually comes next is “take these supplements, avoid these foods, do this workout,” all built on one root cause and a one-size protocol. So what if you don’t fit just one? What if your insulin is high and your stress is off the charts? What if your androgens were high two years ago and have come down? A fixed type can lock you into a plan that doesn’t evolve. I’ve seen someone decide they’re “not insulin-resistant” because they aren’t gaining weight, so they never test it. Or get so afraid of “spiking cortisol” that they avoid the workout they love and would actually do consistently. Once diagnosed, PCOS is an enduring condition, but it isn’t frozen in time. It adapts to what you’re doing, and your care should too.

A better question than “what type am I?”

This is why I don’t use fixed types. Instead I use something that reflects where your body is right now. I call it the PCOS profile, and it’s not a type. It’s a flexible, science-informed way to look at the whole picture, figure out what’s relevant for you right now, and track what we’re watching so changes get noticed before they get loud. It respects real life, that you have constraints and priorities and can’t do everything at once. So if you have no idea which “type” you are, you don’t need to feel bad. And if you feel dogmatic that you’re a certain type, it probably just matched an adaptation that’s working for you, and that’s good feedback. The better question isn’t “what type am I,” it’s “where is my body actually at right now, and what’s worth watching?” That’s the one that gets you somewhere.

Watch the full video: PCOS Types: Why the Labels Don’t Fit, and subscribe to my YouTube channel.

This article is educational, not medical advice or a diagnosis. If something here resonates, bring it to your healthcare team, who can look at it through the lens of your individual health.

Main references

1. Teede HJ, et al. Recommendations from the 2023 International Evidence-based Guideline for the Assessment and Management of Polycystic Ovary Syndrome. J Clin Endocrinol Metab. 2023. PubMed

By Dr. Mélanie DesChâtelets, ND. Licensed naturopathic doctor and founder of PMOS Health Collective.

What if your symptoms look like PMOS (formerly PCOS) but aren’t? A real PCOS diagnosis is two-sided: you have to meet your age-appropriate inclusion criteria, and you have to rule out the look-alikes first. That second half gets skipped all the time. So this is a plain-English guide to the PCOS exclusion labs, the tests that rule out the conditions that mimic PCOS, so you can pull up your own panel and see whether they were actually done. Not to interpret your results, but to ask a sharper question at your next appointment.

One quick note first. This is education, not medical advice, and I’m not your naturopathic doctor. Read it, take notes, then bring your actual panel to your own care team.

▶ Liked this video? Subscribe on YouTube.

Why PCOS is a diagnosis of exclusion

Someone once asked me, “I had polycystic morphology on my ultrasound, isn’t it obvious?” And that’s just not how it works, for good reason. Inclusion criteria are what must be present, and they change with the age you’re diagnosed. Exclusion criteria are what must be absent to avoid a misdiagnosis, and these don’t change with age. They’re the same for everyone. To land a PCOS diagnosis you need both: your age-appropriate inclusion criteria, and a clean set of exclusion labs. So when you read your own file, you’re looking for two halves, not one.

What “mimickers” are

Before anyone calls it PCOS, the other things that look like it have to be ruled out. I call these mimickers, because they produce the same symptoms. Some are common and routinely tested, like thyroid issues and high prolactin. Others are rarer, and those are the ones I see missed. The guidelines are clear that there’s a set of mandatory exclusion labs for everyone, because skipping them means a rare look-alike can be mislabeled as PCOS, and those people deserve to be caught and treated for what they actually have.

The 4 mandatory exclusion labs to look for

There are four, recommended for everyone at diagnosis, regardless of age or how clear the symptoms seem. These are the names to scan your own panel for:

• TSH (thyroid). Thyroid problems can make cycles irregular. If your cycles are off but it’s the thyroid, it might not be PCOS.
• Prolactin. High levels can block ovulation, which can also look like altered cycles.
• 17-OH progesterone. This screens for congenital adrenal hyperplasia, one of the rarer look-alikes.
• FSH. This checks the brain-ovary communication.

If those were done and came back in range, that half of the picture is covered. If something was flagged, that’s where the nuance and follow-up belong, with your provider. Reading your panel here isn’t about deciding what a result means, it’s about confirming the four were even run.

The conditional, case-by-case checks

Then there are conditional ones, ordered case by case. You don’t need these to get a PCOS diagnosis if you’ve met inclusion and cleared the four mandatory labs. But a thoughtful provider keeps them in mind when extra symptoms point elsewhere. A few examples: Cushing’s, if there are features of cortisol excess. Androgen-secreting tumours, if androgen symptoms come on rapidly or severely. And hypothalamic amenorrhea, where a big gap between energy in and energy out (very low body weight, over-exercising, or a stretch of weight loss) leads the body to pause the cycle. So if you don’t see these on your panel, that’s often appropriate. They’re targeted, not routine.

Red flags worth knowing the names of

In PCOS, androgen symptoms are slow and progressive. So sudden or severe hair growth, or voice changes, are red flags for a different cause and deserve a closer look. A round “moon” face, a fat pad at the back of the neck, or purple stretch lines on the abdomen can be red flags for high cortisol and Cushing’s. Very low body weight, an extreme training schedule, or very high stress alongside missing periods can point to hypothalamic amenorrhea. Milk discharge when you’re not breastfeeding, or new headaches and vision changes, can be red flags for a prolactin-related issue. None of these is a diagnosis on its own. They don’t automatically mean something serious is going on, but they can, which is exactly why they’re worth flagging quickly so the right testing gets done.

How to check your own panel

Here’s the empowering part. You can pull up your own bloodwork and see whether the four mandatory labs (TSH, prolactin, 17-OH progesterone, FSH) were actually run during your diagnostic process. This isn’t about interpreting your own results, it’s about walking into your next appointment with a sharper question: “Were the exclusion labs done, and what did they show?” That’s the kind of question that gets you a real answer, and it’s the difference between a label that was applied and a diagnosis that was earned.

Watch the full video: Don’t Call It PCOS Until You’ve Ruled Out These Labs, and subscribe to my YouTube channel.

This article is educational, not medical advice or a diagnosis. If something here resonates, bring it to your healthcare team, who can look at it through the lens of your individual health.

Main references

1. Teede HJ, et al. Recommendations from the 2023 International Evidence-based Guideline for the Assessment and Management of Polycystic Ovary Syndrome. J Clin Endocrinol Metab. 2023. PubMed

By Dr. Mélanie DesChâtelets, ND. Licensed naturopathic doctor and founder of PMOS Health Collective.

If you were diagnosed with PCOS as a teen, there’s something worth knowing: the rules used to diagnose PMOS (formerly PCOS) before age 20 are different from the adult ones, and stricter. A teenage body is still finding its rhythm, so some features that look like PCOS at 15 are just a normal phase. That’s why a label given years ago is worth a second look now, not to second-guess your care, but to make sure you’d actually have met the adolescent criteria in the first place. Here’s how to check.

One quick convention before we start: I’ll use age 20 as the line between the teen rules and the adult rules, just to keep it simple. The real line is about 8 years after your first period. For most of you that lands around 20, for some a little sooner or a little later. So whenever you see 20 in here, read it as shorthand for that 8-year mark.

And one quick note. This is education, not medical advice, and I’m not your naturopathic doctor. Read it, take notes, then bring your history to your own care team.

▶ Liked this video? Subscribe on YouTube.

Why teen diagnosis works differently

In the years right after a first period, the communication between the brain and the ovaries is just getting started. So some things that look like PCOS in a teenager are actually normal for the stage. That’s also why PCOS can look so different from one person to the next, and why a teen label given years ago deserves a recheck. To avoid over-diagnosing during a normal phase, the criteria are stricter before age 20. If you were diagnosed back then, the first thing to know is that the bar was supposed to be higher, not lower.

The adolescent rule: two of two, not two of three

Before age 20, there’s no reliable way to assess ovarian morphology, so ultrasound and AMH are off the table. That turns the adult “two out of three” rule into a stricter “two out of two.” You need both: irregular cycles (defined specifically for teens, below) and androgen signs, either symptoms or bloodwork. If only one of the two was present, that isn’t a full diagnosis. It can be considered “PCOS at risk,” but only once other causes for the symptoms have been explored first. So when you look back at your file, the question is: did I clearly have both?

Reading the androgen box as a teen

The visible signs come first: aggressive acne that doesn’t respond well to treatment or persists as you get older, hair thinning where the part keeps widening, or excess hair on the face tracking toward the belly button or down the legs.

Here’s a clinical pearl worth carrying, especially in hindsight: in PCOS, high-androgen symptoms tend to come on slowly and progressively. If excess facial hair showed up very suddenly and very severely, that can point to other causes and deserves a different workup. So if your symptoms came on fast back then, that’s worth raising clearly with your provider now, so the right testing gets done.

The cycle definitions change by year

This is the part defined differently than for adults, and it hinges on how many years it had been since your first period. In the first year, irregular cycles are normal. From one to under three years out, cycles shorter than 21 days or longer than 45 days count as irregular. At three years and beyond, the window tightens to shorter than 21 or longer than 35. And at any point more than a year out, a single cycle of 90 days or more counts. So “my cycles were irregular as a teen” only means something against the right year-based window. Worth checking which window you were actually in.

Why a teen ultrasound doesn’t count

You may have had a pelvic ultrasound back then for any number of reasons, but for diagnosing PCOS it isn’t reliable until it’s been about 8 years since your first period (which for many lands around age 20). Before that, the ovaries normally show more follicles, so the scan can look “polycystic” when nothing is wrong. So if your teen diagnosis was hanging mostly on an ultrasound, that’s the single biggest reason to revisit it and check whether the other two criteria were actually met.

“PCOS at risk,” and when to reassess

Some teens have a few features but not quite enough for a clear diagnosis: maybe some irregular cycles but little on the androgen side, or the reverse. Once other causes are ruled out, that’s “PCOS at risk.” Not a no, not a yes, a “let’s keep an eye on this.” The recommendation is to reassess once it’s been at least 8 years since the first period (around age 20 for many), when ovarian morphology testing finally becomes accurate, either by ultrasound or by AMH if you’d rather skip an internal scan.

So if you’re carrying a teen label into adulthood, the empowering move is simple: pull your records, see whether you met two of two, and bring it to your provider as a clear question. “I was diagnosed as a teen. Looking at the adolescent criteria, can we confirm I still meet a PCOS diagnosis now?” Whether the answer is a yes, a not-yet, or a watch, knowing exactly where you stand is the whole point, because every decision after it depends on getting this part right.

Watch the full video: Is This Really PCOS? Teen Edition, and subscribe to my YouTube channel.

This article is educational, not medical advice or a diagnosis. If something here resonates, bring it to your healthcare team, who can look at it through the lens of your individual health.

Main references

1. Teede HJ, et al. Recommendations from the 2023 International Evidence-based Guideline for the Assessment and Management of Polycystic Ovary Syndrome. J Clin Endocrinol Metab. 2023. PubMed